Newbie looking for advice…
Hello to all:
I just joined this group and am searching for some help with my SD
(as I recently found out it was called). I have had this problem for
only a year or so (I am 24 years old). For me personally, the
problem consists of mild dryness under the armpit area and sometimes
on my back with itching. However, the biggest problems are (1) my
scalp which constantly flakes and often itches and (2) my face
around my eyebrows, my upper lip, and predominantly on the sides of
my nose and on my face area next to my nose. I have done quite a bit
of research on the topic tonight trying to get a feel for the
condition because it has seemed to get worse for me over the past
few weeks. My skin on my face around my nose is constantly peeling
now and is noticeably red.
I was hoping that someone could give me some starter advice to see
if I can find any basic, easy solutions before moving on to more
drastic measures. From what I have read in my research, it would
appear that my condition is on the mild end of the scale and
subsequently, I am looking for a way to remedy the problem
immediately without having to see a doctor (unless of course it
comes to that in the future). I would really appreciate anyones help
recommending solutions (i.e., scalp treatments, shampoos, anti-yeast
or anti-fungal creams, etc.). As I said, I am truly new at this and
so I only have an idea of some basic treatments from my brief
research. Thanks a bunch in advance!!
June 29th, 2005 at 3:36 pm
I can only help you telling what works for me. That
doesnt mean it will work for you too. Thats isnt how
SD works. But you can always try
After trying various approaches and soaps, i’ve come
up with the following scheme
1) NodeDS shampoo for the scalp every two days
2) lactibon pH 3.5 for the rest of the skin
cheers
———————————
Hello to all:
I just joined this group and am searching for some
help with my SD
(as I recently found out it was called). I have had
this problem for
only a year or so (I am 24 years old). For me
personally, the
problem consists of mild dryness under the armpit area
and sometimes
on my back with itching. However, the biggest problems
are (1) my
scalp which constantly flakes and often itches and (2)
my face
around my eyebrows, my upper lip, and predominantly on
the sides of
my nose and on my face area next to my nose. I have
done quite a bit
of research on the topic tonight trying to get a feel
for the
condition because it has seemed to get worse for me
over the past
few weeks. My skin on my face around my nose is
constantly peeling
now and is noticeably red.
I was hoping that someone could give me some starter
advice to see
if I can find any basic, easy solutions before moving
on to more
drastic measures. From what I have read in my
research, it would
appear that my condition is on the mild end of the
scale and
subsequently, I am looking for a way to remedy the
problem
immediately without having to see a doctor (unless of
course it
comes to that in the future). I would really
appreciate anyones help
recommending solutions (i.e., scalp treatments,
shampoos, anti-yeast
or anti-fungal creams, etc.). As I said, I am truly
new at this and
so I only have an idea of some basic treatments from
my brief
research. Thanks a bunch in advance!!
=====
Running on:
1.5 Ghz P4
256Mb
asus v800x chipset
RH9 CCRMA-patched linux
June 29th, 2005 at 8:08 pm
I think the most effective over the counter stuff for you
may be various anti dandruff shampoos.
These can be made much more than normally effective
by spreading a layer on the skin about 15 to 30 minutes
before bathing.
Some types to try are (look for the active ingredient):
selenium sulfide group (Selsun Blue)
ketoconizole (Nizeral)
pyrithione zinc (Head & Shoulders, DHS Zinc, Zincon)
The last is an extremely broad spectrum antimicrobial agent
(actually, a biocide), but since it kills just about anything,
it can also attack skin and hair folicles with enough exposure.
So I wouldn’t start with a full half hour soak with that one,
till you have some idea how sensitive you are to it
and use it no more often than every other day.
The others can be used daily.
In any case, rinse extra thoroughly because the detergent residue
can also cause irritation.
This does not mean you have to scrub hard.
Just rub gently with your fingers under clean flowing warm water,
a while after it no longer feels soapy.
If you find one that is going to work,
you should see definite improvement in two days or so.
If more than one works, rotate through the successful types,
one a day.
June 30th, 2005 at 10:09 am
there is no immediate safe treatment for seb derm unless you use steroids.
But stay away from them as much as possible. I have been doing excellent
now with hollistic treatments. Im on a regimine of Omega 3’s, Vitamin E,
Detox, Magnesium, Adrenal and on a healthy diet. Never been better.
tony the baloney
June 30th, 2005 at 3:04 pm
Elidel and ciclopiroxolamine(Loprox or Batrafen) creams. They are
effective and safe.
Best wishes
–
August 7th, 2005 at 7:45 am
Hi,
New to group. SD all my life. Was born with the crud. Went away for a
while in childhood only to come back with vengeance in teens.
Your protocol is very similar to mine. I am also getting good
results.
Vit E blocks 5-lipoxygenase pathway(dose dependent), same as boswellia
and that seems to help. I also use Vit C and selenium to boost
glutathione. That is always low in SD. Oxidation load high in SD. CD4
T-cells low. Interferon gamma, IL-2 low. Similar profile to HIV.
The adrenals may be shot and this may be a big clue to a posssible
treatment. Very interesting IMO. This warrants some serious review I
think :
In a similar vein :
August 7th, 2005 at 5:54 pm
Interesting articles,
Scary thing for me was two years ago when the SD spread to my face
and then on to my underarm area, I had no idea what was happening. I
decided a bad rash on my face was not normal and went to my doctor.
He was an old timer late 60s, he examined me, leaned back, looked me
square in the eye as if to tell if my response to his question would
be a lie and asked " Have you ever been at risk for hiv?" Well
needless to say, I shit, broke out in a cold sweat. He explained to
me, that SD is very common in hiv positive people. About 90% get it
and when it spreads from the scalp to other areas of the body, this
is a tale tale sign of being hiv positive or having a bad
functioning immune system.
Well facts are, I had been in a faithful relationship for over 12
years, but hey I’m a guy and prior to this in my younger years there
had been a few ladies. Well the wise old doctor suggested a test.
He said he rarely has ever seen SD anywhere but the scalp or face in
a negative person. He said anytime it is located on the chest area
or other parts of the body, the person’s immune system is in bad
shape usually because of hiv. I was freaked, thought for sure I must
have gotten hiv in my younger years.
Well thank God the test was negative. No hiv for me. All my blood
work came back in the perfect range, shoing my immune function to be
operating perfectly. This indicates to me that this disorder is not
always related to a poor immune function. Has anyone else has this
type of experience?
And as for my progress using vinegar, well it working wonders! My
face is on it’s way and i’m over the hump for sure, past the major
scabs and flaking and moving into smooth skin.
Kevin
August 9th, 2005 at 10:30 am
I thought HIV meds were what caused a lot of them to have SD? Was
your doctor making the claim that having SD(which he said was due to
a week immune system) makes you more susceptible to HIV?
Also, with the vinigar routine, are you following a diet or just
applying and drinking.
Thanks
Ohyes93
August 10th, 2005 at 6:48 am
Oh yes,
No the meds are not the cause of SD, it’s hiv itself. In fact, most
all people with hiv have ongoing problems with SD. Scientists have
discovered that the bodies actual immune reaction to the presence of
hiv is what causes SD to errupt in hiv positive individuals. I.E.,
you’re immune response to the hiv itself is what causes the SD. Most
individuals with hiv will develop SD VERY EARLY after hiv infection
and it will stay with them UNTIL they get treatment. In other words,
SD in hiv positive people happens before their immune system is
damaged! There is so much reading about this on the web you can
spend days researching it! Try Goggle "Seborrheic dermatitis HIV"
You’ll be amazed. SD is the NUMBER #1 early hiv symptom. I hope i’m
not scaring people here, but these are facts. All this being said SD
is prevalent in hiv negative people too.
August 15th, 2005 at 2:19 am
I had Seb, Derm well before having sex, unless my own hand counts
so i know mine isnt related to HIV
tony the baloney the one and only
August 15th, 2005 at 12:28 pm
Hey guys i wanted to give this ACVM treatment a try for my stubborn scalp
problem. Can anyone tell me the step procedures neccessary for this
treatment.
thanks
tony baloney the one and only
August 15th, 2005 at 9:23 pm
ha ha good one, I developed ‘cradle cap’ soon after I was born, which
is just another form of SD so I reckon I’m in the clear too!
Tony, can you be sure that your hand wasn’t seeing anyone else during
that time?
August 16th, 2005 at 7:32 am
Tony baloney,
To do the ACV treatment justice you should really follow the routine
in full as per the recommendations of the ….yadda…..yadda….yadda
If you want to skip ahead and just try ACV on your scalp, this would
probably be an acceptable variation on the routine, but no complaints
please if it doesn’t work because you are technically cheating!
1. Shower twice a day, washing your hair with a mild non-medicated
shampoo, this helps cut through the scale and keep your scalp clean.
2. Dry your hair thoroughly and drench your scalp with 100%
unpasterised, unfiltered, organic, raw apple cider vinegar with the
mother.
3. Leave the vinegar on your scalp, do not wash off. Towel dry hair
or blow dry is ok. Your hair will get all silky smooth and shiney.
4. Repeat for as long as it takes (weeks)
You really are missing out on some very important steps, like a
change in diet, if you only do the things I have mentioned but it’s
worth a try. You should consider what styling products (if any) you
are using on your hair and try to cut back or eliminate. Again,
there is so much more to ACV treatment than I have mentioned, so
please consider doing some research yourself (dare i say at
the ‘other’ group) before you give it try.
People with long time dandruff problems have reported a stop in
itching the very first time they use ACV on their head, please let
the group know what it does for you!
Regards,
Scott no SD problems anymore
August 16th, 2005 at 3:02 pm
I have a question. Why does the ACV work so quick and affective on
the scalp and take much longer to work on the face? I’ve been at
this for 8 days now. My scalp flakes went away almost overnight,
while my face is getting better, but still a mess? Anyone know why
this is? Scalp SD and the stuff on the face is basically the same
thing?
And on a side note, don’t knock the connection between SD and HIV.
Again, it is the most common, frequent symptom of all. If any of you
have had a sudden outburst of SD on the face get tested. Also, if
you have SD anywhere but the face or head, your immune system is
under attack, this is sceintifcally proven. SD on the chest,
underarms, groin, etc, is due to immunity function problems, usually
severe. This is not a joking matter. Ask any doctor if you do not
believe me, or simply look it up on the web. You guys make fun and
don’t get tested. I’m the only here, apparently who got tested!
Kevin
August 17th, 2005 at 3:32 am
ACV treatment is more difficult on the face becasue your facial skin is a
lot more sensitive than scalp skin, this is why I beleive that using full
force acv can be causing a cycle of irritant dermatitis along with the seb
derm–I could tolerate the acv on my face only at 50%, but use it full
strength on scalp without any problems.
Regarding seb derm and HIV–seb derm on a non-HIV person is due to an
over-reactive immune system and is not indicative of an immune system under
attack. This is why immunosuppressants will lessen the seb derm in someone
who is not immuno-compromised.
Those with HIV get seb derm because they cannot fight off the normal levels
of fungus on the skin because their immune system has been compromised.
August 17th, 2005 at 9:29 pm
WRONG!
Nice of you to try and apparently lesson your concern for HIV. It
was a nice try, but here are the facts.
SD usually appears VERY early in HIV infection due to the immune
system being over stimulated and on high speed trying to contain the
virus. It is usally the very FIRST marker, or indication of HIV!!!!
It is the MOST common syptom, do to it’s occurance BEFORE immune
supression!! Most people who experience SD outbreaks and are HIV
positive still have normal T-cell counts! Once again, it’s the
bodies response to the presence of the HIV virus that causes SD. NOT
from a diminished immune system. In the later stages of the disease,
after the immune system has been weakened or destroyed, you will
then see a resurgence in SD flare ups. This can be downright
uncontrollable.
Any unexplained sudden worsening, or onset of middle aged SD (20-
50’s) should warrant the testing for HIV!!! Now, I was a man,
swallowed that hard pill, got tested, maybe some of you should too.
Quit trying to convince yourselves otherwise. If you dipped your
noodle and got it wet, then get tested! I spent over 300 hours
reading this when I had my HIV scare! I know what i’m talking about
here.
August 18th, 2005 at 5:51 am
This very recent study(pub. last month) proves that if
you boost CD4(+) T-cells you clear SD. I suggest we
concentrate on ways to do this.(eg vitamin E, aspirin,
zinc, selenium etc) Note : you can have genetic low
t-cells without HIV. ie I was born with SD in the 60’s
long before AIDS. The approach to treating it is the
same as for HIV IMO. PS : the immune deficiency leads
to gut microflora becoming deranged and acetic acid
and lactic acid producing bacteria are decimated by
pathogens. AVM addresses the acetic acid part of the
equation without the lactic acid part or the lactic
acid bacteria part or the immune system part IMO.
J Invest Dermatol. 2005 Jan;124(1):151-9. Related
Articles, Links
Click here to read
A novel T cell receptor transgenic animal model of
seborrheic dermatitis-like skin disease.
Oble DA, Collett E, Hsieh M, Ambjorn M, Law J,
Dutz J, Teh HS.
Department of Microbiology and Immunology,
University of British Columbia, Vancouver, Canada.
We have characterized a novel animal model of the
common inflammatory skin disease seborrheic dermatitis
(SD) that involves the expression of the self-specific
2C transgenic T cell receptor on the DBA/2 genetic
background. Opportunistic fungal pathogens are present
in the primary histological lesions and severe disease
can be mitigated by the administration of fluconazole,
demonstrating a role for infection in disease
pathogenesis. Spontaneous disease convalescence occurs
at 70-90 d of age and is preceded by an expansion of
CD4(+) T cells that partially restores the T cell
lymphopenia that occurs in these animals. The adoptive
transfer of syngeneic CD4(+) T cells into pre-diseased
DBA/2 2C mice completely abrogates the development of
cutaneous disease. The pattern of disease inheritance
in DBA/2 backcrosses suggests that one, or a closely
linked group of genes, may control disease penetrance.
Bone marrow reconstitution experiments demonstrated
that the DBA/2 susceptibility factor(s) governing
disease penetrance is likely non-hematopoietic since
bone marrow from disease-resistant 2C mice can
adoptively transfer the full disease phenotype to
non-transgenic DBA/2 animals. This model implicates
fungal organisms and CD4(+) T cell lymphopenia in the
development of a SD-like condition and, as such, may
mimic the development of SD in acquired
immunodeficiency syndrome.
August 18th, 2005 at 5:47 pm
could you tell me what AVM is ie "AVM addresses the acetic acid part
of the equation without the lactic acid part or the lactic acid
bacteria part or the immune system part IMO"
August 19th, 2005 at 6:17 am
First, seb derm is not a virus. Second, trying to scare people into thinking
they have HIV because of some redness on their faces is just plain silly.
If your theory is true, then why is seb derm the first indicator of HIV,
why is not a host of other autoimmune related conditions like psoriasis,
asthma, rheumatoid arthirtis, etc–things that are not related to fungus
feeding off of our sweet sebum in our central face and scalp.
Seb derm is simply an over-reaction to normal levels of fungus on our
skin–t-cell counts are silly, it is only the various skin cells in our
skin, not in our blood, that lead to seb derm flares.
Should everyone get tested for HIV, I suppose, I did before I got married-
but I have had seb derm for ages and not once did any of a 6-8 doctors say
that it may be an indicator of HIV.
August 19th, 2005 at 4:26 pm
This study doesn’t prove a thing regarding human adult seb derm and just
about all studies done in the past are polar to the idea that you treat AIDS
and seb derm the same way–I have experimented for years with boosting the
immune system, it doesn’t do shit for seb derm (not that this proves
anything either, but it does for me). On the other hand, everytime I take
prednisone my seb derm vanishes within 1-2 days, and then it always comes
back when I stop take steroid.
Look at all the new biologic drugs on the market and other anti-rejection
drugs for transplant patients–they are dangerous but work for the most part
against autoimmune conditions, seb derm being a relatively weak autoimmune
condition similar to psoriasis more than anything else.
If they do this study on adult humans and not 3 month old mice I would think
diferrently, but in the meantime, I wouldn’t bother going out of your way to
boost the immune system thinking it ia going to help.
August 20th, 2005 at 10:13 am
steve,
i am an artist/musician. my brother is the phd in the family. i get the jist of
what you are saying, but once more in english. based on what you wrote…is
there a to do/don’t do list?
what is your background that you are so comfortable with the technical info?
piotr
August 20th, 2005 at 10:53 pm
I have experimented with low dose prednisone and it did not work, I do not
see full clearance until I hit about 35mgs a day for 2-4 days–I take the
does all at once in the morning with food.
Trying to get the adrenals to naturally produce more cortisone does not seem
to work–if you have any suggestions for this I will attempt them.
I still however do not understand what studies have been done on seb derm in
particular to determine that the immune response is different from
psoriasis, or is this just theory that has not been tested?
I do look at the underlying cause of seb derm with respect to natural
production of GLA (the best source of this is borage oil at 24% GLA, evening
primrose at about 18% GLA is second best), which naturally regulates immune
responses. Studies done on atopics show that many patients had a lazy
enzyme, delta 6 desaturase–this enzyme is responsible for converting LA
into GLA–supplementing with borage oil does help, but it’s long term safety
is questionable and the extraction process of any unregulated supplement is
always questionable as well.
Borage applied directly to skin may help, but not on the face because the
skin is too sensitive and you end up getting contact dermatitis from it or
acne from pores getting clogged.
August 21st, 2005 at 6:41 am
Once again, SD in early stages of HIV is due to an OVERACTIVE IMMUNE
SYSTEM, not a depleted one! The overactive immune system is WHAT
CAUSES SD.
I’m HIV negaitve and have SD, it used to be very bad dandruff and
two years ago when it spread, it was recommended I have an HIV test,
I did, it was negative. For good measure I also had a full CBC
workout and had my lymphocyte subsets done, which is a breakdown of
all you T-cells. My T-4 cells the same cells destroyed by his, the
same cells you claim are low in HIV and SD sufferers, was a whopping
1290!!!!!!!!!!!!!!!!!!!!!! Way above average! I’m as healthy as a
fricken BULL and yet I still have SD!!!
Kevin
August 21st, 2005 at 3:03 pm
35mg prednisone will certainly shut down the adrenals.
Healthy body produces 40mg cortisol per day. Max at
8am and minimum just before bed. 35mg prednisone=175mg
cortisol. Something else to consider is cortisol
resistance(genetic or acquired). This complicates the
picture and blunts cortisol treatment.
TH2 TEND to have insufficient cortisol. Cortisol boost
TH1 cytokines. The diseases of high circulating
cortisol tend to be diseases of the metabolic syndrome
or syndrome X. Heart disease, diabetes type II,
atherosclerosis, psoriasis, some arthritis. The
allergies(asthma eczema rhinitis), CFS, cancer, HIV
tend to be low circulating cortisol. Of course it is
probably not as conveniently simple as this, but it is
the current state of research and a starting point.
At a glance you would think SD belongs in the latter
group. Especially since it is almost always found in
HIV+ at some point in the disease progression. SD
seems dependent on excessive androgens. We should try
and study other diseases of androgen excess and look
for clues in how to treat them. eg polycystic ovary
syndrome(PCOS), acne. I have seen refs where PCOS is
treated with cortisol.
GLA has been found to be ineffective in atopics. The
delta 6 desaturase role has been questioned.
CONCLUSIONS: Supplementation with EFA has no
clinically relevant effect on the severity of AD.
Rather in SD it is the inhibition of 5 alpha-reductase
that seems to be involved and this is tied up with
excess androgens. Cortisol is one inhibitor of this
enzyme, ketoconazole is another popular one, in fact
cortisol has been cited as one treatment for that
other seborrheic disease : acne.
The answer lies in the hormonal system IMO.
A difficult disease.
August 22nd, 2005 at 1:12 am
Here we have some strong suggestion that SD is a TH2
humoral disease :
The study results suggest that Pityrosporum ovale
yeast might be implicated in causing a humoral immune
response, which is important for the developing
inflammatory reactions in Dermatitis seborrhoides
IL-10 is the initiator of the TH2 immune response(and
consequently suppresses TH1) :
However, stimulation of PBMC from SD patients with P.
ovale antigen induced significantly increased IL-10
synthesis. IgA, IgG and IgM synthesis was
significantly increased in cultures of PBMC from
patients with SD whether the cells were
antigen-stimulated or not. Our results support the
assumption that strong skin colonization with P. ovale
in SD is due to an altered cellular immunity, which
may be induced by increased IL-10 secretion.
A subset of atopics also have a similar reaction to
these yeasts. However, so do some psoriatics and that
is where it gets fuzzy……..
August 22nd, 2005 at 3:29 pm
Steve–with regards to EFA, particluarly GLA, it depends on what study you
read–there are several that says GLA supplements help–I did take large
amounts of borage oil to test it and it did work–not full clearance, but a
definite improvment–maybe placebo effect, who knows, but lowering he
amount of borage and my seb derm came back. I hate taking supplements so I
stopped treatment.
I am not convinced, but certainly will keep an open mind, based upon your
theories and studies posted–however, the several synthetic steroid
therapies, prednisone, desowen, elocon, etc or immunomodulators (protopic in
particular) have all worked very sucessfully for me and for a majority for
those who use them–are they are a cure,no–are the side effects worth it,
debatable and a personal decision, but until some human testing is done on
Seb derm specifically with the various therapies you have suggested, it
won’t get acceptance by derms, and thus, we won’t get them prescribed, and
thus, we are stuck with what we have currently.
August 22nd, 2005 at 11:17 pm
So have we come to some type of conclusion here?
August 23rd, 2005 at 9:26 am
My conclusion is that SD in most instances are related to an
overactive immune response to yeast. That the skin is regenerating
at a 20-30% faster rate then in a non SD sufferer. This why SD is so
prevalent in those who have HIV, because their immune system is over
stimulated from that virus, thus causing SD. There is no correlation
between having low T-4 cells and SD in non HIV people. Furthermore,
new scientific studies have indicated the SD in HIV + individuals is
totally different then those without HIV. In HIV + people, the rash
is more red and has less flakes. The rash tends to have more oil,
but not the dead flakes associated with skin destruction.
August 23rd, 2005 at 5:48 pm
Are you saying that in HIV they have an over active
immune system ? That goes against everything I have
ever heard about the disease.
What they do have is an UNDERactive TH1 immune system
and an OVERactive TH2 system. ie they OVERreact to
extracellular yeasts and bacteria and UNDERreact to
intracellular yeasts, bacteria, virus
Here is an overview, and note some treatment
suggestions(albeit unscientific) :
PS : I don’t have the answers, but I am sure the
answers lie in this debate/topic, and I welcome the
debate. After all, we have the same goal.
August 24th, 2005 at 3:57 am
Hi RJ,
I agree that studies can contradict each other. I just
posted that study since it is a study of studies
(meta) and that was the conclusion re FA and GLA. It
is also my experience with these supplements.
I am not sure that we should wait for the derms to get
aggressive. We have the worlds largest library at our
fingertips I reckon we have some serious tracking to
do. I am sure that there are enough clues out there to
crack this. Just need a team effort.
August 25th, 2005 at 12:15 am
Looks like cortisol does block androgens. Antiandrogen
is a proven treatment for SD, this includes
ketoconazole, isotretinoin, steroids. Safe, low dose
cortisol is worth a look IMO.
:
Arch Ital Urol Androl. 2004 Sep;76(3):143-6. Related
Articles, Links
Congenital adrenal hyperplasia.
Spandri A, Cogliardi A, Maggi P, De Giorgi A,
Masperi R, Beccaria L.
Department of Pediatrics, General Hospital
"Alessandro Manzoni", Lecco, Italy.
Congenital adrenal hyperplasia (CAH) is an
autosomal recessive condition characterized by an
insufficient production of cortisol and mineraloactive
hormones with a consequent hyperstimulation of
hypothalamo-pituitary-adrenal feedback and an increase
of androgens. Although the lack of different enzymes
in adrenal steroidogenesis can be responsible for
different forms of the disease, the deficiency of
21-hydroxylase is the more frequent defect. It is
caused by mutations in CYP21 gene located on the short
arm of chromosome 6 and it causes a heterogeneous
phenotype characterized by a classical form (genitalia
virilization in female, early onset acute adrenal
insufficiency with salt wasting, precocious
pseudopuberty, signs of hyperandrogenism), or by a
simple virilizing form (presence of signs of
hyperandrogenism without salt-loosing crises) or by a
non-classic form evidenced only by mild to moderate
signs of hyperandrogenism. The diagnosis can be made
by 17-hydroxyprogesterone measurement in basal and
after ACTH stimulation test. Glucocorticoid and
mineraloactive therapy have been proved to reduce the
risk of adrenal crisis reducing the levels of
androgens and controlling the symptoms of the disease.
August 25th, 2005 at 2:31 pm
The anti-androgen Proscar (propecia) should probably be added to the list as
well–I took 1.25 mg daily of Proscar a few years ago for about 10 months,
it greatly reduced the amount of sebum production as it curbs the natural
production of DHT, which led to less severe seb derm breakouts, but not full
clearance. Side effects were eyebrow thinning and really dry skin, neither
of which I was too fond of so I stopped taking it. Taking the drug at
higher levels could have led to more clearance but I never really
experimented with higher doses.
A good website for proscar and seb derm discussions is www.keratin.com–go
to the forums, the guy you owns this site is a hair loss specialist in
Europe and knows his stuff pretty well when it comes to seb derm, alopecia
areata, psoriasis, etc.
August 25th, 2005 at 8:33 pm
These 3 articles , taken together look like they have
a coherent meaning and could very well apply to us
scalers. They may not, but it is sure suspicious. Note
natural thyroid supplement is said to be a part of the
low-dose cortisol therapy. Whatever the case, I am
foolish enough to want to try it. Lots of reading :
August 27th, 2005 at 2:56 pm
Steve, Kevin,& RJ
I found your recent postings excellent - some of the best
contributions to the group in recent times. Thanks.
I want to take the time to re-read them and will post my humble
comments shortly.
Tom.